OMIM - Lung diseases

Homo sapiens

This study contains curated human phenotype data extracted from OMIM. Additional computational processing and curation of the phenotype data was performed to further organize and group data from related phenotypes into unifying disease categories.

Source: NextBio Library/Mutation and Phenotypic data

Total # Biosets: 8

Caco2 cells infected with ospF mutant S. flexneri

Homo sapiens

Phosphorylation of histone H3 at Serine 10 emerges as a mechanism increasing chromatin accessibility of the transcription factor NF-kB for a particular set of immune genes. Here we report that a bacterial pathogen uses this strategy to shape the transcriptional response of infected host cells. We id…

Authors: Arbibe L, Kim D, Batsche E, Pedron T, Masteescu B, Muchardt C et al.

Organization: Pasteur Institute Biologie cellulaire et…

Total # Biosets: 6