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Schizophrenia is still one of the most mysterious and costliest mental disorders in terms of human suffering and societal expenditure. Here, we focus on the key developments in biology, epidemiology, and pharmacology of schizophrenia and provide a syndromal framework in which these as…
[NEWS] Gene copy number variations of the same region, 16p11.2, are already linked to autism An international team of researchers led by geneticist Jonathan Sebat, Ph.D., of Cold Spring Harbor Laboratory (CSHL), has identified a mutation on human chromosome 16 that substantially incre…
Researchers have identified a mutation on human chromosome 16 that substantially increases risk for schizophrenia.
A researcher has pinpointed the mechanism by which a gene associated with both autism and schizophrenia influences behavior in mice.
A gene called DISC1, (for disrupted in schizophrenia ) has been a leading contender among possible genetic causes since it was implicated in schizophrenia in a large Scottish clan two decades ago. The DISC1 gene codes for a protein important for brain development, as well as for mood …
Scientists studying nerve cells in fruit flies have uncovered a new function for a gene whose human equivalent may play a critical role in schizophrenia.
Researchers say antiepilectic drug treatments administered when the brain is developing appear to trigger schizophrenia-like behaviour in animal models. In humans, having a history of seizures in infancy is a significant risk factor for development of schizophrenia later in life, but …
Northwestern researchers have discovered that schizophrenia symptoms -- which begin to develop in adolescents -- are caused by a low level of a brain protein necessary to build pathways between neurons. Without enough of the protein, there are too few roads for information to flow bet…
Schizophrenia in Greek means "split mind" and is perhaps the most common psychotic disease. As a matter of fact, Schizophrenia is the fourth leading cause of disability in the developed world.Contributor: Arijit ChaudhuryPublished: Aug 19, 2009
An international team of researchers led by geneticist Jonathan Sebat, Ph.D., of Cold Spring Harbor Laboratory (CSHL), has identified a mutation on human chromosome 16 that substantially increases risk for schizophrenia.